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Common pathophysiological mechanisms of metabolic syndrome and NDD include insulin resistance of the brain, neuroinflammation, oxidative stress, dysfunction of the BBB, mitochondrial damage, dysbiosis of the intestinal microbiota and changes in the renin-angiotensin system
It should only be administered under strict medical supervisionideally through an institutional review boardapproved trial
Since in some of the studies analysed type 2 diabetic patients were treated with metformin, which has been shown to enhance GLP-1 responses [51], any potential difference in GLP-1 secretory responses between type 2 diabetic patients and healthy participants may have been modified by this mechanism
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